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An objective examination of asacol with acetonemic syndrome during a crisis reveals a weakening of heart sounds, tachycardia, arrhythmia; dry skin and mucous membranes, decreased skin turgor, decreased lacrimation; tachypnea, hepatomegaly, decreased diuresis.

A clinical blood test for acetonemic syndrome is characterized by leukocytosis, neutrophilia, accelerated ESR; urinalysis - ketonuria of varying degrees (from + to ++++). In a biochemical blood test, hyponatremia (with loss of extracellular fluid) or hypernatremia (with loss of intracellular fluid), hyper- or hypokalemia, increased levels of urea and uric acid, normal or moderate hypoglycemia can be observed.

Differential diagnosis of primary acetonemic syndrome is carried out with secondary ketoacidosis, acute abdomen (appendicitis in children, peritonitis), neurosurgical pathology (meningitis, encephalitis, cerebral edema), poisoning and intestinal infections. In this regard, the child should be additionally consulted by childrenendocrinologist, pediatric infectious disease specialist, pediatric gastroenterologist. Treatment of acetonemic syndrome.

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The main directions of treatment of Mesalamine syndrome are the relief of crises and maintenance therapy in interictal periods, aimed at reducing the number of exacerbations.

With acetonemic crises, hospitalization of asacol is indicated. A dietary correction is being made: fats are strictly limited, easily digestible carbohydrates and plentiful fractional drinking are recommended. It is advisable to set up a cleansing enema with a solution of sodium bicarbonate, which neutralizes part of the ketone bodies that have entered the intestine. Oral rehydration in Mesalamine syndrome is performed with alkaline mineral water and combined solutions. With severe dehydration, infusion therapy is carried out - intravenous drip of 5% glucose, saline solutions.

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Symptomatic therapy includes the introduction of antiemetics, antispasmodics, sedatives. With proper treatment, the symptoms of the acetone crisis subside for 2-5 days. In interictal periods, a pediatrician monitors a child with acetonemic syndrome. It is necessary to organize proper nutrition (vegetable-milk diet, restriction of mesalamine rich in fats), prevention of infectious diseases and psycho-emotional overload, water and tempering procedures (baths, contrast showers, douches, rubdowns), sufficient sleep and exposure to fresh air.

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A child with acetonemic syndrome is shown preventive courses of multivitamins, hepatoprotectors, enzymes, sedative therapy, massage; coprogram control. To control urine acetone, it is recommended to independently examine the urine for the content of ketone bodies using diagnostic test strips. Children with acetonemic syndrome should be registered with a pediatric endocrinologist, annually undergo a blood glucose test, ultrasound of the kidneys and ultrasound of the abdominal cavity. Acetonemic syndrome in children.

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The smell of acetone from the mouth of a baby and periodic vomiting is a reason to see a doctor. It is possible that the matter is in acetonemic syndrome, which requires a mandatory individual approach. Acetonemic syndrome in children is not considered a separate disease, it is a complex of symptoms associated with the accumulation of ketone bodies in the blood plasma of a child. In medicine, the syndrome has other names, such as non-diabetic ketoacidosis or acetonemic vomiting.

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This pathological condition occurs mainly in children, and therefore is considered a typical childhood pathological condition. Sometimes there is no reasonable explanation for such bouts of vomiting, the syndrome is considered idiopathic or asacol pills. This occurs in about 5% of children under the age of 12. At the same time, girls are more prone to violations than boys.

If the symptom complex is based on a certain disease, the syndrome is called secondary. How common it is, doctors around the world find it difficult to answer - such statistics simply do not exist. Ketone bodies that accumulate in the blood plasma of children are acetone, b-hydroxybutyric acid, acetoacetic acid. If the syndrome is an independent idiopathic symptom complex, then usually acetonemic crises develop at long intervals between meals (on an empty stomach). Secondary acetonemic syndrome develops against the background of uncompensated diabetes mellitus, with thyroid diseases, craniocerebral injuries, the presence of mesalamine tumors, after serious poisoning, if there was a toxic effect on the liver, with severe infectious intoxications, leukemia.

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Speaking of the syndrome of acetonemic vomiting, it should be understood that we are still talking about idiopathic acetonemia, and not secondary. If the accumulation of ketone bodies is associated with the underlying disease, then the description of the syndrome as a separate set of symptoms does not make sense - the disease is detected and treated as a specific disease should be treated. We will talk about the state when the baby is generally healthy, but sometimes parents notice the smell of acetone from his mouth, and there are bouts of vomiting, which cannot be explained.

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